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Iconic singer reveals sad diagnosis

<p>Morten Harket, the iconic voice behind A-ha’s global smash "Take on Me", has revealed he has been diagnosed with Parkinson’s disease – a progressive condition that may bring an end to his singing career.</p> <p>The 65-year-old Norwegian singer shared the news in a moving interview published on the band’s official website, explaining that although treatment has eased some of the condition’s physical toll, it has also affected his voice – the very heart of his artistry.</p> <p>“I don’t feel like singing, and for me that’s a sign,” Harket said candidly. “As things stand now, that’s out of the question.”</p> <p>For the past year, Harket has undergone deep brain stimulation, a complex treatment involving two surgeries to implant electrodes on both sides of his brain. The procedure delivers electrical impulses that help control tremors and muscle stiffness, but in Harket’s case, has also dulled his soaring vocal range.</p> <p>Despite the gravity of the diagnosis, Harket said he feels at peace with it. He credits his 94-year-old father for encouraging him to “use whatever works” in coping with the challenges of the degenerative disorder.</p> <p>“There’s so much to weigh up,” he said. “It’s a difficult balancing act between taking the medication and managing its side effects.”</p> <p>A-ha last toured in 2020 and played what may be their final show together in 2022 at the Hollywood Bowl. Harket said he has continued to work on new music but is unsure whether the songs will ever be finished.</p> <p>Still, in classic form, he offered fans words of hope rather than despair.</p> <p>“Don’t worry about me,” he said. “Spend your energy addressing real problems… and know that I am being taken care of.”</p> <p><em>Images: Instagram</em></p>

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The surprising ways that coffee can interfere with medication

<div class="theconversation-article-body"> <p>For many of us, the day doesn’t start until we’ve had our first cup of coffee. It’s comforting, energising, and one of the most widely consumed beverages in the world. But while your morning brew might feel harmless, it can interact with certain medicines in ways that reduce their effectiveness – or increase the risk of side-effects.</p> <p>From common cold tablets to antidepressants, caffeine’s impact on the body goes far beyond a quick energy boost. Tea also contains caffeine but not in the same concentrations as coffee, and doesn’t seem to affect people in the same way. Here’s what you should know about how coffee can interfere with your medications – and how to stay safe.</p> <h2>1. Cold and flu medicines</h2> <p>Caffeine is a stimulant, which means it speeds up the central nervous system. Pseudoephedrine, a decongestant found in cold and flu remedies such as Sudafed, is <a href="https://medlineplus.gov/druginfo/meds/a682619.html">also a stimulant</a>. When taken together, the effects can be amplified – potentially leading to jitters or restlessness, headaches, fast heart rate and insomnia.</p> <p>Many cold medications already contain added caffeine, increasing these risks further. <a href="https://www.mdpi.com/1422-0067/22/10/5146">Some studies</a> also suggest that combining caffeine with pseudoephedrine can raise blood sugar and body temperature – particularly important for people with diabetes.</p> <p>Stimulant effects are also a concern when combining caffeine with ADHD medications such as amphetamines, or with <a href="https://allergyasthmanetwork.org/news/coffee-and-asthma/">asthma drugs</a> such as theophylline, which shares a similar chemical structure to caffeine. Using them together may increase the risk of side-effects such as a rapid heartbeat and sleep disruption.</p> <figure><iframe src="https://www.youtube.com/embed/9eL16Exry48?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <h2>2. Thyroid medication</h2> <p>Levothyroxine, the standard treatment for an underactive thyroid, is highly sensitive to timing – and your morning coffee can get in the way. <a href="https://onlinelibrary.wiley.com/doi/10.1155/2020/7909703">Studies show</a> that drinking coffee too soon after taking levothyroxine can reduce its absorption by up to 50%.</p> <p>Caffeine speeds up gut motility (the movement of food and waste through the digestive tract), giving the drug <a href="https://www.jandonline.org/article/S2212-2672(16)00200-8/abstract">less time to be absorbed</a> – and <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC8002057/#:%7E:text=Several%20studies%20on%20patients%20with,Benvenga%20et%20al.">may also bind</a> to it in the stomach, making it harder for the body to take in. These effects reduce the drug’s bioavailability, meaning less of it reaches your bloodstream where it’s needed. This interaction <a href="https://www.endocrine.org/news-and-advocacy/news-room/2022/drinking-coffee-does-not-hinder-the-absorption-of-liquid-thyroid-medication">is more common</a> with tablet forms of levothyroxine, and less likely with liquid formulations.</p> <p>If absorption is impaired, <a href="https://www.nhs.uk/conditions/underactive-thyroid-hypothyroidism/">symptoms of hypothyroidism</a> – including fatigue, weight gain and constipation – can return, even if you’re taking your medicine correctly.</p> <p>The same timing rule applies to a class of osteoporosis medications called <a href="https://medlineplus.gov/druginfo/meds/a601011.html#precautions">bisphosphonates</a>, including alendronate and risedronate, which also require an empty stomach and around 30-60 minutes before food or drink is taken.</p> <h2>3. Antidepressants and antipsychotics</h2> <p>The interaction between caffeine and mental health medications can be more complex.</p> <p><a href="https://www.nhs.uk/mental-health/talking-therapies-medicine-treatments/medicines-and-psychiatry/ssri-antidepressants/overview/">Selective serotonin reuptake inhibitors</a> (SSRIs), such as sertraline and citalopram, are a type of antidepressant medication <a href="https://purehost.bath.ac.uk/ws/portalfiles/portal/225886346/Lalji_McGrogan_and_Bailey_JADR_2021.pdf">widely used</a> to treat depression, anxiety and other psychiatric conditions. <a href="https://onlinelibrary.wiley.com/doi/10.1155/2020/7909703">Lab studies</a> suggest caffeine can bind to these drugs in the stomach, reducing absorption and potentially making them less effective.</p> <p>Tricyclic antidepressants (TCAs), such as amitriptyline and imipramine, are a class of older antidepressants that work by affecting the levels of neurotransmitters in the brain. They were among the first antidepressants developed and are <a href="https://www.nhs.uk/mental-health/talking-therapies-medicine-treatments/medicines-and-psychiatry/antidepressants/overview/#:%7E:text=Tricyclic%20antidepressants%20(TCAs),to%20treat%20chronic%20nerve%20pain.">less commonly used</a> today, compared with newer antidepressants such as SSRIs, due to their potential for more side-effects and higher risk of overdose.</p> <p>TCAs are broken down by the liver enzyme <a href="https://onlinelibrary.wiley.com/doi/10.1155/2020/7909703">CYP1A2</a>, which also metabolises caffeine. The competition between the two can slow drug breakdown, <a href="https://onlinelibrary.wiley.com/doi/10.1155/2020/7909703">increasing side-effects</a>, or delay caffeine clearance, making you feel jittery or wired longer than usual.</p> <p>Clozapine, an antipsychotic, is also processed by CYP1A2. <a href="https://onlinelibrary.wiley.com/doi/10.1155/2020/7909703">One study showed</a> that drinking two-to-three cups of coffee could increase blood levels of clozapine by up to 97%, <a href="https://medlineplus.gov/druginfo/meds/a691001.html#side-effects">potentially increasing risks</a> such as drowsiness, confusion, or more serious complications.</p> <h2>4. Painkillers</h2> <p>Some over-the-counter painkillers, such as those containing aspirin or paracetamol, include added caffeine. <a href="https://link.springer.com/article/10.2165/00003088-200039020-00004">Coffee can speed up</a> how quickly these drugs are absorbed by accelerating how fast the stomach empties and making the <a href="https://onlinelibrary.wiley.com/doi/10.1155/2020/7909703">stomach more acidic</a>, which improves absorption for some medications such as aspirin.</p> <p>While this may help painkillers work faster, it could also raise the risk of side-effects like stomach irritation or bleeding, especially when combined with other sources of caffeine. Though no serious cases have been reported, caution is still advised.</p> <h2>5. Heart medications</h2> <p>Caffeine can temporarily raise blood pressure and heart rate, typically lasting three-to-four hours after consumption. For people taking blood pressure medication or drugs that control irregular heart rhythms (arrhythmias), this <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC8101832/#:%7E:text=Table%20I.&amp;text=The%20next%20stage%20of%20hypertension,response%20to%20calcium%20channel%20blockers.&amp;text=The%20potential%20for%20caffeine%20to,Table%20II%20summarizes%20these%20recommendations.">may counteract</a> the intended effects of the medication.</p> <p>This doesn’t mean people with heart conditions must avoid coffee altogether – but they should monitor how it affects their symptoms, and consider limiting intake or switching to decaf if needed.</p> <figure><iframe src="https://www.youtube.com/embed/r-YwCCNDOy0?wmode=transparent&amp;start=0" width="440" height="260" frameborder="0" allowfullscreen="allowfullscreen"></iframe></figure> <h2>What can you do?</h2> <p>Coffee may be part of your daily routine, but it’s also a potent chemical compound that can influence how your body processes medicine. Here’s how to make sure it doesn’t interfere.</p> <p>Take levothyroxine or bisphosphonates on an empty stomach with water, and wait 30-60 minutes before drinking coffee or eating breakfast.</p> <p>Be cautious with cold and flu remedies, asthma treatments and ADHD medications, as caffeine can amplify side-effects.</p> <p>If you’re on antidepressants, antipsychotics, or blood pressure drugs, discuss your caffeine habits with your doctor.</p> <p>Consider reducing intake or choosing a decaffeinated option if you experience side-effects like restlessness, insomnia or heart palpitations.</p> <p>Everyone metabolises caffeine differently – some people feel fine after three cups, while others get side-effects after just one. Pay attention to how your body responds and talk to your pharmacist or GP if anything feels off.</p> <p>If you’re ever unsure whether your medicine and your coffee are a good match, ask your pharmacist or doctor. A short conversation might save you weeks of side-effects or reduced treatment effectiveness – and help you enjoy your brew with peace of mind.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/256919/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/dipa-kamdar-1485027">Dipa Kamdar</a>, Senior Lecturer in Pharmacy Practice, <a href="https://theconversation.com/institutions/kingston-university-949">Kingston University</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/coffee-can-interfere-with-your-medication-heres-what-you-need-to-know-256919">original article</a>.</em></p> <p><em>Image: Pexels / Jonathan Borba</em></p> </div>

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Why do some people need less sleep than others?

<div class="theconversation-article-body"> <p>Have you ever noticed how some people bounce out of bed after just a few hours of sleep, while others can barely function without a solid eight hours?</p> <p>Take Margaret Thatcher, for example. The former British prime minister was known for sleeping <a href="https://www.bbc.com/news/magazine-22084671">just four hours a night</a>. She worked late, rose early, and seemed to thrive on little sleep.</p> <p>But for most of us, that kind of sleep schedule would be disastrous. We’d be groggy, unfocused, and reaching for sugary snacks and caffeinated drinks by mid-morning.</p> <p>So why do some people seem to need less sleep than others? It’s a question that’s fascinated scientists for years. Here’s what we know so far.</p> <h2>Natural short sleepers</h2> <p>There is a small group of people who don’t need much sleep. We call them <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC6879540/">natural short sleepers</a>. They can function perfectly well on just four to six hours of sleep each night, often for their entire lives.</p> <p>Generally they <a href="https://academic.oup.com/sleep/article/44/Supplement_2/A154/6260529">don’t feel tired</a>, they don’t nap, and they don’t suffer the usual negative consequences of sleep deprivation. Scientists call this the natural short sleep phenotype – a biological trait that allows people to get all the benefits of sleep in less time.</p> <p>In 2010 researchers discovered genetic mutations that help explain this phenomenon. Natural short sleepers carry rare variants <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC2884988/">in certain genes</a>, which seem to make their sleep more efficient.</p> <p>More recently, a <a href="https://www.pnas.org/doi/epub/10.1073/pnas.2500356122">2025 study</a> assessed a woman in her 70s with one of these rare mutations. Despite sleeping just six hours a night for most of her life, she remained physically healthy, mentally sharp, and led a full, active life. Her body, it seems, was simply wired to need less sleep.</p> <p>We’re still learning about how common these genetic mutations are and why they occur.</p> <h2>Not everyone who sleeps less is a natural short sleeper</h2> <p>But here’s the catch: most people who think they’re natural short sleepers aren’t. They’re just chronically sleep-deprived. Often, their short sleep is due to long work hours, social commitments, or a belief sleeping less is a sign of strength or productivity.</p> <p>In today’s hustle culture, it’s common to hear people boast about getting by on only a few hours of sleep. But for the average person, that’s not sustainable.</p> <p>The effects of short sleep build up over time, creating what’s known as a “sleep debt”. This <a href="https://www.sciencedirect.com/science/article/pii/S0149763417301641">can lead to</a> poor concentration, mood swings, micro-sleeps (brief lapses into sleep), reduced performance and even <a href="https://www.sciencedirect.com/science/article/pii/S1389945716301381">long-term health risks</a>. For example, short sleep has been linked to an increased risk of obesity, diabetes, high blood pressure and cardiovascular disease (heart disease and stroke).</p> <h2>The weekend catch-up dilemma</h2> <p>To make up for lost sleep during the week, many people try to “catch up” on weekends.</p> <p>This can help repay some of the sleep debt that has accumulated in the short term. Research <a href="https://www.sciencedirect.com/science/article/pii/S2352721823001663?via%3Dihub">suggests</a> getting one to two extra hours of sleep on the weekend or taking naps when possible may help reduce the negative effects of short sleep.</p> <p>However, it’s not a perfect fix. Weekend catch-up sleep and naps may not fully resolve sleep debt. The topic remains one of ongoing scientific debate.</p> <p>A recent <a href="https://academic.oup.com/sleep/article/47/11/zsae135/7696120">large study</a> suggested weekend catch-up sleep may not offset the cardiovascular risks associated with chronic short sleep.</p> <p>What’s more, large swings in sleep timing can disrupt your body’s <a href="https://www.nature.com/articles/s41598-017-03171-4">internal clock</a>, and sleeping in too much on weekends may make it harder to fall asleep on Sunday night, which can mean starting the working week less rested.</p> <p>Increasing evidence indicates <a href="https://academic.oup.com/sleep/article/47/1/zsad253/7280269">repeated cycles of irregular sleep</a> may have an important influence on general health and the risk of early death, potentially even more so than how long we sleep for.</p> <p>Ultimately, while moderate catch-up sleep might offer some benefits, it’s no substitute for consistent, high-quality sleep throughout the week. That said, maintaining such regularity can be particularly challenging for people with non-traditional schedules, such as shift workers.</p> <h2>So, was Thatcher a true natural short sleeper?</h2> <p>It’s hard to say. Some reports suggest <a href="https://www.theguardian.com/lifeandstyle/2021/aug/27/from-aristotle-to-einstein-a-brief-history-of-power-nappers">she napped during the day</a> in the back of a car between meetings. That could mean she was simply sleep-deprived and compensating for an accumulated sleep debt when she could.</p> <p>Separate to whether someone is a natural short sleeper, there are a range of other reasons people may need more or less sleep than others. Factors <a href="https://www.sleephealthfoundation.org.au/sleep-topics/how-much-sleep-do-you-really-need">such as age</a> and underlying health conditions can significantly influence sleep requirements.</p> <p>For example, older adults often experience changes in their circadian rhythms and are more likely to suffer from fragmented sleep due to conditions <a href="https://link.springer.com/article/10.1007/s00702-019-02067-z">such as arthritis</a> or <a href="https://link.springer.com/article/10.1007/s11886-023-01939-x">cardiovascular disease</a>.</p> <p>Sleep needs vary from person to person, and while a lucky few can thrive on less, most of us need <a href="https://www.sleephealthfoundation.org.au/sleep-topics/how-much-sleep-do-you-really-need">seven to nine hours</a> a night to feel and function our best. If you’re regularly skimping on sleep and relying on weekends to catch up, it might be time to rethink your routine. After all, sleep isn’t a luxury – it’s a biological necessity.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/256342/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/kelly-sansom-2390567">Kelly Sansom</a>, Research Associate, College of Medicine and Public Health, Flinders University; Research Associate, Centre for Healthy Ageing, <a href="https://theconversation.com/institutions/murdoch-university-746">Murdoch University</a> and <a href="https://theconversation.com/profiles/peter-eastwood-2316718">Peter Eastwood</a>, Deputy Vice Chancellor, Research and Innovation, <a href="https://theconversation.com/institutions/murdoch-university-746">Murdoch University</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/why-do-some-people-need-less-sleep-than-others-a-gene-variation-could-have-something-to-do-with-it-256342">original article</a>.</em></p> <p><em>Image: </em></p> </div>

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"Three months to live": Channel 10 star shares emotional battle with incurable cancer

<p>Channel 10 presenter Barry Du Bois has opened up in a raw and heartbreaking letter about his ongoing battle with cancer, revealing the devastating moment he was told he had just months to live.</p> <p>In a deeply personal essay for <a href="https://www.goldcoastbulletin.com.au/entertainment/celebrity-life/channel-10-star-barry-du-bois-reveals-harrowing-moment-he-was-given-three-months-to-live/news-story/20a2e0a910358c1861d2c4d23c8cd617" target="_blank" rel="noopener">The Gold Coast Bulletin</a>, the father of two recalled the chilling day a doctor delivered a prognosis that shattered his world: “I had three months to live.”</p> <p>“I was sitting in a cold, unfamiliar consult room at the hospital, my wife’s hand holding mine … then a doctor who had known me for only a few hours looked me in the eye and told me I had three months to live,” Du Bois wrote.</p> <p>Du Bois was first diagnosed in 2010 with solitary plasmacytoma, a rare and aggressive form of blood cancer. While he survived that initial battle, the disease returned in 2017 – this time as multiple myeloma, an incurable cancer that attacks the immune system and bone marrow.</p> <p>Since then, the 63-year-old <em>Living Room</em> star has fought on, defying the odds with remarkable strength and grace. But as he revealed in the letter, his path has been anything but easy.</p> <p>His strength, he said, was forged through years of pain: breaking his back in a 14-metre fall, a gruelling and heartbreaking IVF journey with his wife Leonie that ended in miscarriage, and her own cervical cancer diagnosis just weeks later.</p> <p>“When I got my diagnosis – incurable cancer, three months to live – I didn’t fall apart,” he wrote. “I knew that from leaning into the previous adversities of life I had the resilience to give the fight of my life.”</p> <p>Still, Du Bois admitted there was a time he came perilously close to giving up. After Leonie’s miscarriage and illness, the darkness nearly swallowed him. “I avoided conversation and started a continual negative conversation with myself that took me into the darkness … depression is a lonely state and I refused to share my pain. I saw it as a weakness.”</p> <p>In the end, it was his family that pulled him back and helped him find purpose again. Since then, Du Bois has become a powerful voice for others fighting chronic illness and mental health struggles, using his platform to share candid updates on life, treatment and the importance of hope.</p> <p>“I was overwhelmed with fear, uncertainty and the unknown,” he said of that early diagnosis. “But through it all, I realised something that I feel is why I am here today: It wasn’t going to be cancer that defined me but the way I choose to approach it.”</p> <p>From his early TV days on <em>The Renovators</em> in 2011 to his long-running role on <em>The Living Room</em> alongside Amanda Keller, Miguel Maestre and Dr Chris Brown, Du Bois has become a familiar and much-loved face on Australian screens. But behind the smiles and strength lies a story of extraordinary pain and courage.</p> <p>His fight continues. And so too does his mission: to live fully, love deeply, and face each day not with fear, but with determination.</p> <p><em>Images: Instagram</em></p>

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World's oldest practicing doctor shares his secrets to a long life

<p>While most people slow down with age, Dr Howard Tucker has spent more than a century proving that passion and purpose are the best medicine.</p> <p>At 102 years old, the American neurologist has lived a life so extraordinary it earned him a Guinness World Record as the oldest practicing doctor – an honour he received just before turning 99. Though he officially hung up his white coat at 100, Tucker remains as active and engaged as ever.</p> <p>Today, he lectures future physicians at Case Western Reserve University in Cleveland, Ohio, and consults on medico-legal cases – work made possible by a law degree he earned at 67. His message? Retirement is not a requirement.</p> <p>“Retirement, I think, is the enemy of longevity,” Dr Tucker recently told Al Roker on NBC’s <em>Today</em>. “You have to have some purpose in life and get up in the morning and know what you’re about.”</p> <p>Even after the hospital where he worked shut its doors in 2022, ending his medical practice at 100, Tucker hasn’t ruled out returning to clinical work. “If it wasn’t for the closure, I’d absolutely still be seeing patients,” he told <em>People</em>. “Nobody wants me at my age – but I’ll keep trying.”</p> <p>His enthusiasm for life has captured the public imagination, especially through <em>What’s Next</em>, a documentary about his life produced by his grandson. The film has gone viral on TikTok, turning the centenarian into an unexpected social media sensation.</p> <p>Dr Tucker credits his longevity not just to good genes – his parents lived to 84 and 96 – but to lifestyle choices. “Heredity and family history of longevity is a healthy start,” he wrote in his Guinness World Record submission. “However, it must be supported by moderation of nutrition, alcohol and happiness.”</p> <p>His daily routine reflects that philosophy. He snowshoes in the winter, walks on the treadmill for at least four kilometres a day, and eats a mostly clean diet – fruit and cereal in the morning, fish and vegetables at night. Lunch is often skipped to maintain mental clarity. Dessert, however, is a staple – typically fruit or ice-cream, enjoyed with his wife of over 70 years.</p> <p>And while he rarely drinks, he allows himself the occasional martini. One thing he’s always avoided: cigarettes.</p> <p>Dr Tucker also says its important to keep the brain stimulated. “If they retire from their work, they should at least do something as a hobby,” he told <em>Today</em>. “You need a stimulus for the brain daily.”</p> <p>Science backs him up. Research shows that ongoing learning, social connection, and a sense of purpose contribute to mental acuity and longevity. Tucker’s life is a blueprint for all three. He remains close to his four children and 10 grandchildren and is an avid sports fan.</p> <p>Despite a fall in his late 80s that ended his skiing days with a broken neck, Tucker hasn’t let injury slow him down. Instead, he’s adapted, always finding new ways to stay active.</p> <p>With his 103rd birthday on the horizon in July, Dr Tucker says he isn’t concerned about the end. “I never think of death,” he said. “To be alive is to know that you’re going to die because life is a fatal disease. And so I live it.”</p> <p>For a man who’s devoted his life to healing others, Dr Howard Tucker’s greatest lesson might be how to truly live.</p> <p><em>Images: Instagram</em></p>

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What’s the difference between ageing and frailty?

<div class="theconversation-article-body"> <p>Ageing is a normal part of the life course. It doesn’t matter how many green smoothies you drink, or how many “anti-ageing” skin care products you use, you can’t stop the <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC2685272/">ageing process</a>.</p> <p>But while we’re all getting older, not everyone who ages will necessarily become frail. Ageing and frailty are closely related, but they’re not the same thing.</p> <p>Let’s break down the difference between the two.</p> <h2>What is ageing?</h2> <p>On a biological level, ageing is the result of the build-up of <a href="https://www.who.int/news-room/fact-sheets/detail/ageing-and-health">cellular and molecular damage</a> in the body over time.</p> <p>The ageing process causes a gradual decline in physical and mental function, a higher risk of disease, and eventual (and unavoidable) death.</p> <p>Still, some people think they can cheat the system, <a href="https://fortune.com/well/article/bryan-johnson-live-longer-unrecognizable-anti-aging-procedure/">spending millions</a> trying to stay young forever. While we may be able to reduce the <a href="https://theconversation.com/do-these-three-popular-anti-ageing-skincare-ingredients-work-heres-what-the-evidence-says-182200">appearance of ageing</a>, ultimately there’s <a href="https://www.nature.com/articles/s44324-024-00040-3">no magic pill</a> to <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC2685272/">increase our longevity</a>.</p> <p>Around one in six Australians are over the age of 65 (<a href="https://www.aihw.gov.au/reports/older-people/older-australians/contents/demographic-profile">16% of the total population</a>). Yet <a href="https://www.tandfonline.com/doi/full/10.1080/03601277.2024.2402056">as individuals</a> and <a href="https://theconversation.com/fear-of-ageing-is-really-a-fear-of-the-unknown-and-modern-society-is-making-things-worse-220925">a society</a> many of us still have a <a href="https://www.smh.com.au/lifestyle/health-and-wellness/we-ve-been-constructed-to-think-a-certain-way-the-psychology-of-ageing-20231213-p5er6a.html">fear of ageing</a>.</p> <p>But what is it about ageing we are so afraid of? When it comes down to it, many people are probably less afraid of ageing, and more afraid of becoming frail.</p> <h2>What is frailty?</h2> <p><a href="https://www.afn.org.au/what-is-frailty/">Frailty</a> is defined as a state of vulnerability characterised by a loss of reserve across multiple parts of the body.</p> <p>Frailty is generally characterised by <a href="https://www1.racgp.org.au/newsgp/clinical/frailty-declared-a-medical-condition">several physical symptoms</a>, such as weakness, slow walking speed, exhaustion, unintentional weight loss, and low activity level.</p> <p>Lower bone density and osteoporosis (a condition where the bones become weak and brittle) are also <a href="https://josr-online.biomedcentral.com/articles/10.1186/s13018-024-04875-w">associated with frailty</a>, increasing the risk of <a href="https://www.sciencedirect.com/science/article/pii/S1279770723020250">falls and fractures</a>.</p> <p>Notably, someone who is frail is less able to “bounce back” (or recover) after a stressor event compared to someone who is not frail. A stressor event could be, for example, having a fall, getting a urinary infection, or even being admitted to hospital.</p> <p>Frailty is more common in older people. But in some cases, frailty can affect younger people too. For example, people with advanced chronic diseases, such as <a href="https://academic.oup.com/eurjcn/article/22/4/345/6775229">heart failure</a>, can <a href="https://onlinelibrary.wiley.com/doi/full/10.5694/mja15.00801">develop frailty</a> much younger.</p> <p>Frailty is dynamic. While it can get worse over time, in some cases <a href="https://www.sciencedirect.com/science/article/pii/S037851221830478X">frailty can also be reversed</a> or even prevented through health and lifestyle changes.</p> <p>For example, we know physical inactivity and a sedentary lifestyle can <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(19)31786-6/abstract">significantly increase a person’s risk</a> of becoming frail. On the flip side, evidence shows doing more exercise can <a href="https://pubmed.ncbi.nlm.nih.gov/36746389/">reduce frailty in older adults</a>.</p> <p>There are other lifestyle modifications we can make too. And the earlier we make these changes, the better.</p> <h2>Preventing frailty</h2> <p>Here are some <a href="https://youtu.be/41cMkvsaOOM">key things</a> you can do to <a href="https://www.self.com/story/how-to-avoid-frailty-old-age">help prevent frailty</a>:</p> <p><strong>1. Get moving</strong></p> <p>Exercise more, including resistance training (such as squats and lunges, or grab some stretchy resistance bands). Many of these sorts of exercises can be done at home. YouTube has some <a href="https://youtu.be/XDQo4wslr7I?si=FAoyHLDZgSG5AN1r">great resources</a>.</p> <p>You might also consider joining a gym, or asking your GP about seeing an accredited exercise physiologist or physiotherapist. Medicare <a href="https://www9.health.gov.au/mbs/fullDisplay.cfm?type=item&amp;q=10953&amp;qt=item">subsidies may be available</a> for these specialists.</p> <p>The <a href="https://www.health.gov.au/topics/physical-activity-and-exercise/physical-activity-and-exercise-guidelines-for-all-australians/for-older-australians-65-years-and-over">physical activity guidelines</a> for older Australians recommend at least 30 minutes of moderate intensity physical activity on most days or preferably every day.</p> <p>The guidelines also highlight the importance of incorporating different types of activities (such as resistance, balance or flexibility exercises) and reducing the time you spend sitting down.</p> <p><strong>2. Stay socially active</strong></p> <p>Social isolation and loneliness can <a href="https://academic.oup.com/gerontologist/article-abstract/64/10/gnae114/7734069">contribute to the progression of frailty</a>. Reach out to friends and family for support or contact local community groups that you may be able to join. This might include your local Zumba class or bridge club.</p> <p><strong>3. Ask your doctor or pharmacist to regularly check your medications</strong></p> <p>“Polypharmacy” (when someone is prescribed <a href="https://www.safetyandquality.gov.au/our-work/healthcare-variation/fourth-atlas-2021/medicines-use-older-people/61-polypharmacy-75-years-and-over">five or more medications</a>) is associated with an increased <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC6005607/">risk of frailty</a>. The presence of frailty can also interfere with how the <a href="https://www.sciencedirect.com/science/article/pii/S0047637419300387">body absorbs medicines</a>.</p> <p><a href="https://www.healthdirect.gov.au/home-medicines-review">Home medicine reviews</a> are available for older adults with a <a href="https://www9.health.gov.au/mbs/fullDisplay.cfm?type=item&amp;q=900">chronic medical condition or a complex medication regimen</a>. These reviews aims to help people get the most benefit from their medicines and reduce their risk of <a href="https://www.safetyandquality.gov.au/sites/default/files/2021-04/fourth_atlas_2021_-_6.2_medications_management_reviews_75_years_and_over_0.pdf">experiencing adverse effects</a>.</p> <p>Always consult your doctor before making any changes to your current medications.</p> <p><strong>4. Eat a protein-rich diet with plenty of fruit and vegetables</strong></p> <p><a href="https://academic.oup.com/biomedgerontology/article/61/6/589/589472?login=true#9578331">Low nutrient intake</a> can negatively impact physical function and may increase your risk of becoming frail. There’s some evidence to suggest eating more protein may <a href="https://academic.oup.com/ageing/article/49/1/32/5618813">delay the onset of frailty</a>.</p> <p>A food-first approach is best when looking to increase the <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC7598653/#:%7E:text=Many%20studies%20have%20described%20an,are%20necessary%20to%20prevent%20frailty.">protein in your diet</a>. Protein is found in <a href="https://www.betterhealth.vic.gov.au/health/healthyliving/protein#protein-foods">foods such as</a> lean meats, poultry, seafood, eggs, dairy products, legumes and nuts.</p> <p>Adults over 50 should aim to eat <a href="https://www.eatforhealth.gov.au/nutrient-reference-values/nutrients/protein">64 grams of protein</a> per day for men and 46g per day for women. Adults over 70 should aim for 81g per day for men and 57g per day for women.</p> <p>Ask your GP for a referral to a dietitian who can provide advice on a dietary regime that is best for you.</p> <p>Supplements may be recommended if you are struggling to meet your protein needs from diet alone.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/247450/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/julee-mcdonagh-1525476">Julee McDonagh</a>, Senior Research Fellow of Frailty Research, <a href="https://theconversation.com/institutions/university-of-wollongong-711">University of Wollongong</a> and <a href="https://theconversation.com/profiles/caleb-ferguson-72">Caleb Ferguson</a>, Professor of Nursing and Director of Health Innovations, <a href="https://theconversation.com/institutions/university-of-wollongong-711">University of Wollongong</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/whats-the-difference-between-ageing-and-frailty-one-is-inevitable-the-other-is-not-247450">original article</a>.</em></p> <p><em>Image: Mikhail Nikov / Pexels</em></p> </div>

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"Have some decency": Appalling reaction to Magda's health news

<p>Australian comedian and national treasure Magda Szubanski has revealed her battle with a rare and aggressive blood cancer – only to be met with a wave of online vitriol and conspiracy theories, drawing widespread condemnation for the lack of compassion.</p> <p>On Thursday, the 64-year-old star <a href="https://www.oversixty.com.au/health/caring/magda-szubanski-shares-devastating-health-news" target="_blank" rel="noopener">announced she had been diagnosed with Mantle Cell Lymphoma</a>, a rare form of stage-4 blood cancer. The diagnosis came unexpectedly during a routine breast screening, prompting Szubanski to begin the “Nordic protocol”, which she described as “one of the best treatments available”.</p> <p>In a candid Instagram video, the <em>Kath & Kim</em> actress addressed her followers with honesty and humour. “Hello, my lovelies. The head is shaved in anticipation of it all falling out in a couple of weeks,” she said. “It’s pretty confronting… But new treatments keep coming down the pipeline all the time.”</p> <p>Despite the seriousness of her condition, Szubanski reassured fans that she was in good hands, supported by her loved ones and an exceptional medical team. “To be honest, I’ve been feeling pretty ratsh*t for ages. So I asked for extra bloods and – voila!”</p> <p>However, the warmth of her message was quickly overshadowed by a torrent of cruel remarks and baseless conspiracy theories on social media platform X (formerly Twitter). Some users linked her cancer diagnosis to her vocal support for COVID-19 vaccinations during the pandemic, reviving the discredited “turbo cancer” myth – a fringe theory suggesting COVID vaccines cause rapid-onset cancers.</p> <p>“Very sad to hear another experimental vaccine pusher has been diagnosed with something horrible,” one user tweeted. Others echoed similar sentiments, with one post reading, “Zero sympathy,” and another claiming, “It’s good to see that karma still works.”</p> <p>Szubanski was a prominent proponent of vaccination during the pandemic, even donning her beloved character Sharon Strzelecki in a government campaign to encourage Victorians to get the jab. That effort, once celebrated, is now being weaponised by some online as supposed justification for her illness.</p> <p>The backlash sparked swift condemnation from more compassionate voices on the platform. “As if cancer didn’t exist before Covid and vaccinations. You guys are seriously cooked,” wrote one user. Another added, “Classless of you to post this. Why drop to the level?”</p> <p>Many others pointed out the lack of basic human decency in mocking someone for a life-threatening diagnosis. “The posts here are highly disrespectful… Have some decency. BTW, these things can happen to anyone.”</p> <p>Szubanski’s diagnosis has drawn an outpouring of support from fans, friends, and fellow celebrities who praised her courage and vulnerability in going public. As she begins treatment, the beloved entertainer remains focused on her recovery and grateful for the support surrounding her.</p> <p>“I’ll be lying very low while my immune system takes a hammering,” she said. “For now, just know I’m in good hands, good spirits – but I reserve my human right to be a cranky old moll.”</p> <p><em>Image: Supplied</em></p>

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Magda Szubanski shares devastating health news

<p>Beloved actor and comedian Magda Szubanski has revealed she is facing a deeply personal and harrowing health battle, after being diagnosed with a rare and aggressive form of cancer.</p> <p>The 64-year-old star, best known for her iconic roles in <em>Kath & Kim</em>, <em>Babe</em>, and <em>Ride Like a Girl</em>, shared the devastating news in an emotional message posted to social media on Wednesday.</p> <p>"Hello, my lovelies," she began, her trademark warmth shining through despite the gravity of her words. "The head is shaved in anticipation of it all falling out in a couple of weeks."</p> <p>Szubanski went on to reveal that she has been diagnosed with a “very rare, very aggressive lymphoma”, which she described as “one of the nasty ones unfortunately”.</p> <p>The news has rocked fans across the country, many of whom have followed Szubanski's decades-long career and have embraced her not only as a performer, but as a passionate advocate for LGBTQ+ rights and mental health awareness.</p> <p>Despite the confronting nature of her diagnosis, Szubanski offered a glimpse of her enduring spirit. “It’s pretty confronting. It is a full-on one,” she said. “But new treatments keep coming down the pipeline all the time … I’ve just got to (laughs). What do you? What are you gonna do?”</p> <p>In the post, she expressed gratitude for the people around her, noting, “The good thing is I’m surrounded by beautiful friends and family and an incredible medical support team. Honestly, we have the best in the world here in Australia.”</p> <p>She also asked fans and well-wishers to keep their distance physically due to her compromised immune system: “Please keep a distance though because I will be very immunocompromised. So as I can’t hug no more.”</p> <p>The actor indicated that more information will be shared in an official statement soon.</p> <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/reel/DKOM_tZSuO2/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/reel/DKOM_tZSuO2/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Magda Szubanski (@magda_szubanski)</a></p> </div> </blockquote> <p>Szubanski has long been open about her private struggles. In her acclaimed 2014 memoir <em>Reckoning</em>, she candidly wrote about her complex relationship with food, identity, and her sexuality. Her public coming out in 2012 was a landmark moment, one she later described as among the most frightening experiences of her life.</p> <p>Messages of love and support have flooded social media, with fans, fellow actors and public figures rallying around one of Australia’s most cherished cultural voices.</p> <p><em>Image: Instagram</em></p>

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What makes somebody a narcissist?

<div class="theconversation-article-body"> <p>Narcissism has become the armchair diagnosis of the decade. Social media is awash with people flinging the label around. Everyone’s ex seems to be a narcissist, some of our parents are under suspicion, and that office villain? They definitely tick the box, too.</p> <p>The accuracy of these rampant diagnoses warrants scepticism. But the reality is narcissists do exist. At its extreme, narcissism is a rare mental health diagnosis, known as <a href="https://www.ncbi.nlm.nih.gov/books/NBK556001/">narcissistic personality disorder</a>. But narcissism also describes a cluster of personality traits, which we all display to varying degrees.</p> <p>For those of us who have been in close quarters with someone high in narcissistic traits, we rarely walk away unscathed. And we may be left with lingering questions. For example, what made them this way?</p> <p>In a recent <a href="https://doi.org/10.1016/j.paid.2025.113255">meta-analysis</a>, my colleagues and I pulled together studies examining the link between narcissism and adult attachment styles. Our findings offer an important clue – especially when it comes to the potential roots of vulnerable narcissism.</p> <h2>Types of narcissism</h2> <p>There are two main types of <a href="https://doi.org/10.1111/j.1467-6494.2010.00711.x">narcissism</a>.</p> <p>Grandiose narcissism is what typically comes to mind. It is characterised by an overtly grandiose, aggressive and dominant interpersonal style. In contrast, vulnerable narcissism is marked by introversion, hypersensitivity to criticism, and a defensive, insecure grandiosity that masks fragile self-esteem.</p> <p>Antagonistic traits such as entitlement, manipulation, and a lack of empathy lie at the core of both narcissism types. This helps to explain the <a href="https://doi.org/10.1037/abn0000298">interpersonal difficulties</a> linked to each.</p> <p>Vulnerable narcissism, in particular, has been linked to a range of harmful behaviours in romantic relationships. Individuals high in this trait are more likely to engage in <a href="https://www.respectvictoria.vic.gov.au/news/red-flags-what-love-bombing-and-why-it-bad">love bombing</a>, <a href="https://doi.org/10.1016/j.tele.2023.101985">ghosting and breadcrumbing</a>.</p> <p>They also tend to report <a href="https://doi.org/10.1080/15298868.2019.1707272">lower relationship satisfaction</a>, hold more permissive attitudes towards <a href="https://doi.org/10.1037/fam0001126">infidelity</a> and perpetrate <a href="https://doi.org/10.1177/15248380231196115">intimate partner violence</a> at higher rates.</p> <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/reel/DIyj175t6iV/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/reel/DIyj175t6iV/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Amanda E. White, LPC, LMHC (@therapyforwomen)</a></p> </div> </blockquote> <h2>Secure versus insecure attachment</h2> <p>Researchers have turned to <a href="https://theconversation.com/attachment-theory-what-people-get-wrong-about-pop-psychologys-latest-trend-for-explaining-relationships-195034">attachment styles</a> to help explain how individuals high in narcissism behave in romantic relationships.</p> <p><a href="https://doi.org/10.1002/9781118938119.ch2">Attachment theory</a> proposes that early experiences with primary caregivers shape our beliefs about ourselves and others. These beliefs are thought to persist into adulthood and influence how we experience and navigate adult relationships.</p> <p>If we felt safe, loved and supported as children, we are more likely to have a positive view of our self and others. This is the hallmark of secure attachment, which lays the foundation for healthy, stable relationships in adulthood.</p> <p>But when early relationships are marked by neglect, inconsistency or abuse, they can give rise to insecure attachment styles. <a href="https://psycnet.apa.org/record/1991-33075-001">Adult attachment models</a> generally identify three types of insecure attachment.</p> <p>Preoccupied attachment develops from a negative view of the self and a positive view of others. Individuals with this style often feel unworthy of love and seek constant reassurance in relationships, fearing rejection and abandonment.</p> <p>Dismissive attachment is rooted in a positive view of the self but a negative view of others. These individuals tend to prioritise independence over intimacy. As a result, they often struggle to form deep connections.</p> <p>Fearful attachment involves negative views of both the self and others. Those with this style typically crave connection while at the same time fearing it, leading to push-pull dynamics in relationships.</p> <h2>An interesting pattern</h2> <p>In our meta-analysis, we combined the results of 33 previous studies comprising more than 10,000 participants to examine how narcissism relates to each of the four adult attachment styles. Overall, narcissism was linked to each of the three insecure attachment styles.</p> <p>But when we looked at the two types of narcissism separately, an interesting pattern emerged. Vulnerable narcissism was consistently linked to insecure attachment styles – with associations of moderate strength for preoccupied and fearful attachment styles.</p> <p>In contrast, grandiose narcissism showed no such link.</p> <p>Does this mean insecure attachment causes vulnerable narcissism? Not necessarily. The studies we reviewed were “correlational”, which means they looked at connections, not causes. So we can’t say attachment styles cause vulnerable narcissism. To answer that, we’d need longitudinal research tracking people over time.</p> <p>Still, our findings suggest that insecure attachment – particularly preoccupied and fearful attachment styles – may be an important risk factor in the development of vulnerable narcissism.</p> <p>Of course, not everyone with an insecure attachment style has high levels of vulnerable narcissism. However, for some, vulnerable narcissism may emerge as a defensive coping strategy that arises when early attachments were marked by inconsistency, neglect or abuse.</p> <h2>Healing childhood wounds</h2> <p>Attachment styles tend to be <a href="https://doi.org/10.1207/S15327957PSPR0602_03">fairly consistent throughout a person’s life</a>, however change is possible. Attachment-focused therapies, such as <a href="https://theconversation.com/im-a-failure-how-schema-therapy-tackles-the-deep-rooted-beliefs-that-affect-our-mental-health-250789">schema therapy</a> and <a href="https://www.psychologytoday.com/au/therapy-types/emotionally-focused-therapy">emotionally focussed therapy</a>, can help individuals heal attachment wounds and build more secure relationship patterns. These approaches may be especially helpful for those high in vulnerable narcissism.</p> <p>At the same time, it is important that families have access to free and timely mental health care, so that children are supported to process and heal from trauma before it shapes their adult relationships, and the way they parent the next generation.</p> <p>But prevention is better than cure.</p> <p>Supporting parents and caregivers to build secure attachments with the their children and equipping them with the tools to parent effectively is essential. This is especially urgent given disturbingly <a href="https://onlinelibrary.wiley.com/doi/full/10.5694/mja2.51873">high rates of child maltreatment in Australia</a>, including emotional abuse, physical abuse and neglect – all of which have been <a href="https://onlinelibrary.wiley.com/doi/full/10.5694/mja2.51873">linked to the development of vulnerable narcissism</a>.</p> <p>We don’t need to look too far to see the cost of turning a blind eye.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/257468/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/megan-willis-8914">Megan Willis</a>, Associate Professor, School of Behavioural and Health Sciences, <a href="https://theconversation.com/institutions/australian-catholic-university-747">Australian Catholic University</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/what-makes-somebody-a-narcissist-mounting-evidence-suggests-links-to-insecure-attachment-styles-257468">original article</a>. </em></p> <p>Images: <span style="font-family: 'Canva Sans', 'Helvetica Neue', Roboto, -apple-system, blinkmacsystemfont, sans-serif; font-size: 14px; white-space: pre;">Engin Akyurt / pexels.com</span></p> </div>

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"Haven’t got long to live": Beloved Harry Potter star's sad health update

<p>Veteran actress Miriam Margolyes, best known to many as Professor Sprout in the Harry Potter franchise, has spoken movingly about her deteriorating health and approaching mortality, revealing she likely has only a few years left to live.</p> <p>In a recent interview with <em>The Times</em>, the 84-year-old star discussed her decision to step away from the big screen, admitting that her body is no longer strong enough to perform the roles she once loved.</p> <p>“When you know that you haven’t got long to live – and I’m probably going to die within the next five or six years, if not before – I’m loathe to leave behind performing,” she said. “It’s such a joy. I yearn to play roles that don’t confine me to wheelchairs, but I’m just not strong enough.”</p> <p>Margolyes has been increasingly open about her health challenges in recent years. In 2023, during an appearance on the Table Manners podcast, she revealed she had undergone heart surgery, <a href="https://www.oversixty.com.au/health/caring/aussie-harry-potter-star-undergoes-heart-surgery" target="_blank" rel="noopener">receiving a cow’s aortic</a> valve to replace her own. “I don’t know how common it is. I’d never heard of that operation,” she said. “But it saves you from having open-heart surgery, which would be infinitely more invasive.”</p> <p>She later told <em>British Vogue</em> that her perspective on death had changed with age. “When you’re young, you never think about death. You just think about your next f**k, basically,” she joked. “I think about death a lot. You can’t help but be aware that the amount of time ahead is less than the time before you.”</p> <p>Despite the seriousness of her condition, Margolyes maintains her trademark wit and philosophical outlook. “I’m still ducking and diving. I’m still open to new experiences. I’m just very conscious that there is no light at the end of the tunnel.”</p> <p>In 2024, Margolyes publicly disclosed she is living with spinal stenosis, a painful and debilitating condition that has severely impacted her mobility. “I’m registered disabled. I use all kinds of assistance,” she shared with <em>Closer Magazine</em>. “I’ve got two sticks and a walker and they’re such a bore, but I’ve just got a mobility scooter, which is a lot of fun.”</p> <p>The <em>Age of Innocence</em> actress also acknowledged her financial worries, saying her biggest fear is outliving her resources. Yet, through it all, she remains strong, funny and honest – qualities that have endeared her to fans for decades.</p> <p><em>Images: Instagram</em></p>

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Amanda Keller breaks down in deeply moving on-air moment

<p>In an emotional and deeply moving on-air moment, beloved broadcaster Amanda Keller broke down in tears while marking her 35th wedding anniversary with husband Harley Oliver – a celebration tinged with sadness as the couple continues to navigate his battle with Parkinson’s disease.</p> <p>Speaking candidly on JAM Nation with Jonesy & Amanda, Keller reflected on the gravity of the milestone and the shifting realities of a marriage tested by illness and time. </p> <p>“I don’t want to get emotional,” she began, her voice trembling. “It’s my wedding anniversary today. And in the old days, Harley and I would have been going out to dinner tonight. But he’s not well enough to do that.”</p> <p>Keller, who has long been a staple of Australian radio and television, spoke of the moment so many couples take for granted: saying vows filled with promise, unaware of the storms that may lie ahead.</p> <p>“When you stand there on your wedding day and you say forever, what does that even mean?” she asked aloud. “You don’t even know.”</p> <p>Oliver was diagnosed with Parkinson’s in 2017, but the couple kept the news private until Keller spoke publicly about his condition in 2023 on her Double A Chattery podcast. She recounted the subtle early signs – his dragging leg, trembling hands – and the devastating confirmation of what they feared most: a degenerative neurological disorder with no cure.</p> <p>“In sickness and in health. And yet that’s where we are. And no concept of what that means until you’re living it,” Keller said. “I kind of fluctuate between taking great pride in the fight that we are alongside each other, and the strength that it takes to get up every day and still fight it and still live it.”</p> <p>“You don’t get to cherry pick life,” Keller continued. “If you’re living life, the sands shift beneath your feet. But we’re still in it together. And I’m grateful for that.”</p> <p>Despite her gratitude, Keller didn’t hide the hardship, admitting she sometimes has the “absolute sh*ts” with their reality. “It’s hard,” she said plainly.</p> <p>Still, there was a glimmer of joy. The couple’s sons would be joining them for dinner – a different kind of celebration, quieter but no less full of love. “We’ve lived a rich and wonderful life together, and we still do,” Keller admitted. “But it’s not the same as the old days.</p> <p>"Anyway, happy anniversary, Harley.”</p> <p>Keller closed with a reflection on the unpredictability of life and love – and the unbreakable bond forged through years of shared struggle and strength. “We all think that we’ll get to 90 and die in our sleep,” she said. “But people face stuff every day. That’s the meat of life. That’s the meat of a long-term relationship. So, I’m grateful to have that.”</p> <p><em>Images: Instagram</em></p>

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TGA approves new drug for Alzheimer’s

<div class="theconversation-article-body"> <p>This week, Australia’s <a href="https://www.tga.gov.au/resources/artg/420194">Therapeutic Goods Administration</a> (TGA) approved a drug called donanemab for people in the early stages of Alzheimer’s disease.</p> <p>Donanemab has previously been approved in a number of other countries, including <a href="https://www.fda.gov/drugs/news-events-human-drugs/fda-approves-treatment-adults-alzheimers-disease">the United States</a>.</p> <p>So what is donanemab, and who will be able to access it in Australia?</p> <blockquote class="twitter-tweet"> <p dir="ltr" lang="en">A drug that is given once a month to slow the cognitive decline associated with Alzheimer’s disease has been approved by the TGA, but will only be available for a select few who can afford it privately. <a href="https://t.co/sJqR3IKhAB">https://t.co/sJqR3IKhAB</a></p> <p>— ABC News (@abcnews) <a href="https://twitter.com/abcnews/status/1925378845135274425?ref_src=twsrc%5Etfw">May 22, 2025</a></p></blockquote> <h2>How does donanemab work?</h2> <p>There are more than 100 different causes <a href="https://www.alzint.org/about/dementia-facts-figures/types-of-dementia/">of dementia</a>, but Alzheimer’s disease alone accounts for about 70% of these, making it <a href="https://www.alzint.org/about/dementia-facts-figures/types-of-dementia/alzheimers-disease/">the most common form</a> of dementia.</p> <p>The disease is believed to be caused by the accumulation in the brain of two abnormal proteins, amyloid and tau. The first is thought to be particularly important, and the “<a href="https://theconversation.com/alzheimers-drug-donanemab-has-been-hailed-as-a-turning-point-for-treatment-but-what-does-it-mean-for-people-with-the-disease-209970">amyloid hypothesis</a>” – which suggests amyloid is the key cause of Alzheimer’s disease – has driven research for many years.</p> <p>Donanemab is a “monoclonal antibody” treatment. Antibodies are proteins the immune system produces that bind to harmful foreign “invaders” in the body, or targets. A monoclonal antibody has one specific target. In the case of donanemab it’s the amyloid protein. Donanemab binds to amyloid protein deposits (plaques) in the brain and allows our bodies to remove them.</p> <p>Donanemab is given monthly, via intravenous infusion.</p> <h2>What does the evidence say?</h2> <p>Australia’s approval of donanemab comes as a result of a <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC10352931/">clinical trial</a> involving 1,736 people published in 2023.</p> <p>This trial showed donanemab resulted in a significant slowing of disease progression in a group of patients who had either early Alzheimer’s disease, or mild cognitive impairment with signs of Alzheimer’s pathology. Before entering the trial, all patients had the presence of amyloid protein detected via <a href="https://www.healthdirect.gov.au/pet-scan">PET scanning</a>.</p> <p>Participants were randomised, and half received donanemab, while the other half received a placebo, over 18 months.</p> <p>For those who received the active drug, their Alzheimer’s disease progressed 35% more slowly over 18 months compared to those who were given the placebo. The researchers ascertained this using the <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC4806404/">Integrated Alzheimer’s Disease Rating Scale</a>, which measures cognition and function.</p> <p>Those who received donanemab also demonstrated large reductions in the levels of amyloid in the brain (as measured by PET scans). The majority, by the end of the trial, were considered to be below the threshold that would normally indicate the presence of Alzheimer’s disease.</p> <p>These results certainly seem to vindicate the amyloid hypothesis, which had been <a href="https://pubmed.ncbi.nlm.nih.gov/33049114/">called into question</a> by the results of multiple <a href="https://www.wsj.com/articles/eli-lillys-alzheimers-drug-trial-fails-to-achieve-goals-1479902563">failed previous studies</a>. They represent a major advance in our understanding of the disease.</p> <p>That said, patients in the study did not <em>improve</em> in terms of cognition or function. They continued to decline, albeit at a significantly slower rate than those who were not treated.</p> <p>The actual clinical significance has been <a href="https://academic.oup.com/braincomms/article/6/6/fcae435/7915707">a topic of debate</a>. Some experts have questioned whether the meaningfulness of this result to the patient is worth the potential risks.</p> <h2>Is the drug safe?</h2> <p>Some 24% of trial participants receiving the drug experienced brain swelling. The rates rose to 40.6% in those possessing two copies of a gene called ApoE4.</p> <p>Although three-quarters of people who developed brain swelling experienced no symptoms from this, there were three deaths in the treatment group during the study related to donanemab, likely a result of brain swelling.</p> <p>These risks <a href="https://www.sciencedirect.com/science/article/pii/S2274580725000949">require regular monitoring</a> with MRI scans while the drug is being given.</p> <p>Some 26.8% of those who received donanemab also experienced small bleeds into the brain (microhaemorrhages) compared to 12.5% of those taking the placebo.</p> <h2>Cost is a barrier</h2> <p>Reports indicate donanemab could cost anywhere between <a href="https://www.abc.net.au/news/2025-05-22/alzheimers-drug-donanemab-tga-approval-dementia/105319856">A$40,000 and $80,000</a> each year in Australia. This puts it beyond the reach of many who might benefit from it.</p> <p>Eli Lilly, the manufacturer of donanemab, has made an application for the drug to be listed on the Pharmaceutical Benefits Scheme, with a decision pending perhaps within <a href="https://www.abc.net.au/news/2025-05-22/alzheimers-drug-donanemab-tga-approval-dementia/105319856">a couple of months</a>. While this would make the drug substantially more affordable for patients, it will represent a large cost to taxpayers.</p> <p>The cost of the drug is in addition to costs associated with the monitoring required to ensure its safety and efficacy (such as doctor visits, MRIs and PET scans).</p> <h2>Who will be able to access it?</h2> <p>This drug is only of benefit for people with early Alzheimer’s-type dementia, so not everybody with Alzheimer’s disease will get access to it.</p> <p>Almost 80% of people who were screened to participate <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC10352931/">in the trial</a> were found unsuitable to proceed.</p> <p>The terms of the <a href="https://www.ebs.tga.gov.au/servlet/xmlmillr6?dbid=ebs/PublicHTML/pdfStore.nsf&amp;docid=420194&amp;agid=%28PrintDetailsPublic%29&amp;actionid=1">TGA approval</a> specify potential patients will first need to be found to have specific levels of amyloid protein in their brains. This would be ascertained either by PET scanning or by lumbar puncture sampling of spinal fluid.</p> <p>Also, patients with two copies of the ApoE4 gene have been ruled unsuitable to receive the drug. The TGA has judged the risk/benefit profile for this group to be unfavourable. This genetic profile accounts for only 2% of the general population, but <a href="https://www.nih.gov/news-events/nih-research-matters/study-defines-major-genetic-form-alzheimer-s-disease">15% of people with Alzheimer’s disease</a>.</p> <h2>Improving diagnosis and tempering expectations</h2> <p>It’s estimated more than <a href="https://www.aihw.gov.au/reports/dementia/dementia-in-aus/contents/population-health-impacts-of-dementia/prevalence-of-dementia">400,000 Australians</a> have dementia. But only <a href="https://www.aihw.gov.au/reports/dementia/ndap-indicators-dashboard/contents/action-4/measure-4-5-time-from-symptom-onset-to-diagnosis">13% of people</a> with dementia currently receive a diagnosis within a year of developing symptoms.</p> <p>Given those with very early disease stand to benefit most from this treatment, we need to expand our dementia diagnostic services significantly.</p> <p>Finally, expectations need to be tempered about what this drug can reasonably achieve. It’s important to be mindful this is not a cure.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/257321/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/steve-macfarlane-4722">Steve Macfarlane</a>, Head of Clinical Services, Dementia Support Australia, &amp; Associate Professor of Psychiatry, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/the-tga-has-approved-donanemab-for-alzheimers-disease-how-does-this-drug-work-and-who-will-be-able-to-access-it-257321">original article</a>.</em></p> </div>

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Could cold sores increase the risk of Alzheimer’s disease?

<div class="theconversation-article-body"> <p>A <a href="https://bmjopen.bmj.com/content/15/5/e093946">new study</a> has found the herpes simplex virus type 1 (HSV-1), which causes cold sores, may be linked to the development of Alzheimer’s disease.</p> <p>This idea is not entirely new. Previous research has suggested there <a href="https://onlinelibrary.wiley.com/doi/10.1002/jmv.1890330403">may be an association</a> between HSV-1 and Alzheimer’s disease, the <a href="https://www.who.int/news-room/fact-sheets/detail/dementia">most common form of dementia</a>.</p> <p>So what can we make of these new findings? And how strong is this link? Let’s take a look at the evidence.</p> <h2>First, what is HSV-1?</h2> <p>HSV-1 is a neurotropic virus, meaning it can infect nerve cells, which send and receive messages to and from the brain. It’s an extremely common virus. The <a href="https://www.who.int/news-room/fact-sheets/detail/herpes-simplex-virus">World Health Organization estimates</a> nearly two-thirds of the global population aged under 50 carries this virus, often unknowingly.</p> <p>An initial infection can cause mild to severe symptoms including fever, headache and muscle aches, and may manifest as blisters and ulcers around the mouth or lips.</p> <p>After this, HSV-1 typically <a href="https://pmc.ncbi.nlm.nih.gov/articles/PMC8923070/">lies dormant</a> in the body’s nervous system, sometimes reactivating due to stress or illness. During reactivation, it can cause symptoms such as cold sores, although in many people it doesn’t cause any symptoms.</p> <h2>What did the new research look at?</h2> <p>In a study published this week <a href="https://bmjopen.bmj.com/content/15/5/e093946">in BMJ Open</a>, researchers analysed data from hundreds of thousands of people drawn from a large United States health insurance dataset.</p> <p>They conducted a matched “case-control” analysis involving more than 340,000 adults aged 50 and older diagnosed with Alzheimer’s disease between 2006 and 2021. Each Alzheimer’s disease patient (a “case”) was matched to a control without a diagnosis of Alzheimer’s disease based on factors such as age, sex and geographic region, a method designed to reduce statistical bias.</p> <p>The team then examined how many of these people had a prior diagnosis of HSV-1 and whether they had been prescribed antiviral treatment for the infection.</p> <p>Among people with Alzheimer’s disease, 0.44% had a previous HSV-1 diagnosis, compared to 0.24% of controls. This translates to an 80% increased relative risk of Alzheimer’s disease in those diagnosed with HSV-1, however the absolute numbers are small.</p> <p>The researchers also found people who received antiviral treatment for HSV-1 had roughly a 17% lower risk of developing Alzheimer’s disease compared to those who were untreated.</p> <h2>Not a new hypothesis</h2> <p>This isn’t the first time researchers have speculated about a viral role in Alzheimer’s disease. <a href="https://onlinelibrary.wiley.com/doi/10.1002/jmv.1890330403">Earlier studies</a> have detected <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(96)10149-5/abstract">HSV-1 DNA in postmortem brain tissues</a> from people who had Alzheimer’s disease.</p> <p>Laboratory research has also shown <a href="https://doi.org/10.1016/j.neuron.2018.06.030">HSV-1 can trigger amyloid-beta plaque accumulation</a> in nerve cells and mouse brains. Amyloid-beta plaques are one of the defining features of Alzheimer’s disease pathology, so this has led to speculation that reactivation of the virus may contribute to brain inflammation or damage.</p> <p>But importantly, previous research and the current study show associations, not proof HSV-1 causes Alzheimer’s disease. These links do not confirm the virus initiates or drives disease progression.</p> <blockquote class="bluesky-embed" data-bluesky-uri="at://did:plc:f3ph3ymzcnhpchwqiu4u37g2/app.bsky.feed.post/3lpn5axtbvj2i" data-bluesky-cid="bafyreifwdab7rnvdpu6t3vnupdymct77ojzrhjkwxcndc2lo5b4ipjkvee" data-bluesky-embed-color-mode="system"> <p lang="">Got Cold Sores? You Might Be at Higher Risk for Alzheimer’s https://gizmodo.com/got-cold-sores-you-might-be-at-higher-risk-for-alzheimers-2000603873</p> <p><a href="https://bsky.app/profile/did:plc:f3ph3ymzcnhpchwqiu4u37g2/post/3lpn5axtbvj2i?ref_src=embed">[image or embed]</a></p> <p>— Gizmodo (<a href="https://bsky.app/profile/did:plc:f3ph3ymzcnhpchwqiu4u37g2?ref_src=embed">@gizmodo.com</a>) <a href="https://bsky.app/profile/did:plc:f3ph3ymzcnhpchwqiu4u37g2/post/3lpn5axtbvj2i?ref_src=embed">May 21, 2025 at 8:31 AM</a></p></blockquote> <h2>Some other important caveats</h2> <p>The study relied on insurance claim data, which may not always reflect accurate or timely clinical diagnoses. HSV-1 is also frequently underdiagnosed, especially when symptoms are mild or absent. These points could explain why both the Alzheimer’s group and the control group saw such low rates of HSV-1, when population rates of this virus <a href="https://www.who.int/news-room/fact-sheets/detail/herpes-simplex-virus">are estimated to be far higher</a>.</p> <p>This means many carriers of HSV-1 in the study may have gone unrecorded and therefore makes the link harder to interpret clearly. The dataset also doesn’t capture how often people had recurring symptoms, or the severity or duration of infections – conditions which might influence risk more directly.</p> <p>Another complicating factor is people with HSV-1 might differ in other ways from those without it. Differences in health-care access, the health of a person’s <a href="https://www.thelancet.com/journals/laninf/article/PIIS1473-3099(21)00144-4/fulltext">immune system</a>, <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(24)01296-0/abstract">lifestyle</a>, <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(13)60630-3/abstract">genetics</a>, or even <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(24)01296-0/abstract">education</a> – could all influence Alzheimer’s disease risk.</p> <h2>So should you be concerned if you have cold sores?</h2> <p>The short answer is no – at least not based on current evidence. Most people with HSV-1 will never develop Alzheimer’s disease. The vast majority live with the virus without any serious neurological issues.</p> <p>The “herpes hypothesis” of Alzheimer’s disease is an interesting area for further research, but far from settled science. This study adds weight to the conversation but doesn’t offer a definitive answer.</p> <p>Alzheimer’s disease is a complex condition with <a href="https://www.thelancet.com/commissions-do/dementia-prevention-intervention-and-care">multiple risk factors</a>, including age, genetics, heart health, education, lifestyle and environmental exposures.</p> <p>Infections such as HSV-1 may be one part of a larger, interconnected puzzle, but they are highly unlikely to be the sole cause.</p> <p>With this in mind, the best thing to do is to focus on what we already know can help keep your brain healthy as you age. <a href="https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(25)00184-9/abstract#:%7E:text=Emerging%20evidence%20suggests%20that%20sedentary,impairment%20and%20reduce%20dementia%20risk.">Regular physical activity</a>, <a href="https://www.health.harvard.edu/blog/sleep-well-and-reduce-your-risk-of-dementia-and-death-2021050322508">good quality sleep</a>, <a href="https://www.alzheimers.org.uk/about-dementia/managing-the-risk-of-dementia/reduce-your-risk-of-dementia/social-isolation">social engagement</a>, <a href="https://www.alzheimersresearchuk.org/dementia-information/dementia-risk/diet-and-dementia-risk/">a balanced diet</a> and <a href="https://www.health.harvard.edu/mind-and-mood/protect-your-brain-from-stress">managing stress</a> can all support long-term brain health.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/257140/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/joyce-siette-1377445">Joyce Siette</a>, Associate Professor | Deputy Director, The MARCS Institute for Brain, Behaviour, and Development, <a href="https://theconversation.com/institutions/western-sydney-university-1092">Western Sydney University</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/could-cold-sores-increase-the-risk-of-alzheimers-disease-a-new-study-is-no-cause-for-panic-257140">original article</a>.</em></p> <p><em>Image: Gizmodo</em></p> </div>

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Brave boxer dies at 25 just two weeks after getting married

<p>The boxing world is in mourning following the heartbreaking news that Georgia O’Connor, a rising star and beloved figure in British sport, has passed away at just 25 after a courageous battle with a rare and aggressive form of cancer.</p> <p>O’Connor, who once stood atop the podium with Commonwealth Youth gold in 2017 and later turned professional with a flawless 3-0 record, revealed her cancer diagnosis earlier this year. She had also been living with ulcerative colitis, a chronic inflammatory bowel disease, showing extraordinary strength both in and out of the ring.</p> <p>Despite being surrounded by support from fans and fellow fighters alike, the Durham-born athlete died earlier this week, leaving the sporting community reeling with grief.</p> <p>In her final post on Instagram, made just two weeks before her passing, Georgia shared a radiant photo of her wedding ring, announcing that she had married her partner Adriano. “The day I married the love of my life,” she wrote.</p> <p>In February, she penned a heartfelt tribute to her husband, calling him her “superhero” and “real-life prince”. Her words captured the depth of their bond during her illness: “From the moment I was diagnosed with cancer, Adriano didn’t hesitate. He quit his job without a second thought and made it his mission to fight this battle alongside me… I have never known love like this.”</p> <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/p/DJjF1RCMnlo/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/DJjF1RCMnlo/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Georgia Cardinali (@georgiaoconnor_1)</a></p> </div> </blockquote> <p>Tributes have since poured in from across the sporting world. Boxing promoter BOXXER, who represented O’Connor, said: “We are heartbroken by the passing of Georgia O’Connor. A true warrior inside and outside the ring, the boxing community has lost a talented, courageous and determined young woman far too soon.”</p> <p>Ben Shalom, BOXXER’s founder, added: “It’s hard to make sense of this. Georgia was an inspirational person and one I’ll never forget. My heart goes out to her parents and partner.”</p> <p>Frank Warren’s Queensberry Promotions shared their condolences, writing: “Everyone at Queensberry Promotions are deeply saddened… Rest in peace, Georgia.”</p> <p>England Boxing also paid tribute, highlighting her decorated amateur career: “A gifted boxer and beloved member of the amateur boxing community, she won medals at the Commonwealth Youth Games, Youth Worlds & European Championships.”</p> <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/p/DFfnk84IZb5/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/DFfnk84IZb5/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Georgia Cardinali (@georgiaoconnor_1)</a></p> </div> </blockquote> <p>O’Connor's talents were not confined to the ring. A three-time national taekwondo champion and undefeated kickboxer, she was also musically gifted, sharing videos of herself singing and playing guitar with fans online. YouTuber-turned-boxer Jake Paul responded simply but powerfully: “F*** cancer. RIP Georgia.”</p> <p>Fellow boxer Joe Laws posted a photo with her, captioning it: “Fighter till the end. Rest easy bro.”</p> <p>Georgia O’Connor's life was short, but her impact was profound. In every arena she entered – whether in gloves or with a guitar – she brought strength, grace and authenticity.  She is survived by her husband Adriano, her family and countless fans who will never forget her spirit.</p> <p><em>Images: Instagram</em></p>

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Patti Newton hospitalised

<p>Entertainment icon Patti Newton has revealed she is recovering in hospital following major surgery. The beloved 80-year-old star took to Instagram to share an update with fans, posting a heartwarming photo of herself in a hospital bed while embracing her grandson, Alby. In the caption, Newton confirmed she had undergone a full hip replacement.</p> <p>“Had a full hip replacement and feeling fine. Thank you to all at Epworth hospital and my wonderful surgeon,” she wrote. “I’ll be chasing Alby around again in no time.”</p> <p>The surgery comes just months after Newton was forced to temporarily step back from her role in <em>Grease: The Musical</em> due to health issues. In January, she announced on Instagram that she would be missing preview performances in Sydney after contracting Covid-19.</p> <p>“So sorry to be missing the previews for <em>Grease</em>,” she wrote at the time, alongside a photo of herself dressed as her character Miss Lynch. “Unfortunately Covid has hit me again. Looking forward to being back next week.”</p> <p>Newton also contracted Covid in December 2023, just as she was preparing to take the stage in the Melbourne season of the hit musical. Despite the setback, she remained optimistic, posting a message to fans on New Year’s Day.</p> <p>“Happy New Year, hope it’s a wonderful 2024. Covid has hit me but wishing the cast of Grease an amazing first show. See you back there soon,” she wrote, sharing a photo of her legs wrapped in a blanket.</p> <p>Newton’s positive attitude continues to inspire fans. As she recovers from surgery, many have taken to social media to send well wishes and messages of support.</p> <p><em>Image: Instagram</em></p>

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Can you treat headaches with physiotherapy?

<div class="theconversation-article-body">You might’ve noticed some physiotherapists advertise they offer treatments for headaches and wondered: would that work?</p> <p>In fact, there’s a solid body of research showing that physiotherapy treatments can be really helpful for certain types of headache.</p> <p>Sometimes, however, medical management is also necessary and it’s worth seeing a doctor. Here’s what you need to know.</p> <h2>Cervicogenic headache: when pain travels up your neck</h2> <p>Cervicogenic headache is where pain is referred from the top of the neck (an area known as the upper cervical spine).</p> <p>Pain is usually one-sided. It generally starts just beneath the skull at the top of the neck, spreading into the back of the head and sometimes into the back of the eye.</p> <p>Neck pain and headache are often triggered by activities that put strain on the neck, such as holding one posture or position for a long time, or doing repetitive neck movements (such as looking up and down repeatedly).</p> <p>Unlike in migraine, people experiencing cervicogenic headache don’t usually get nausea or sensitivity to light and sound.</p> <p>Because this is a musculoskeletal condition of the upper neck, physiotherapy treatments that <a href="https://pubmed.ncbi.nlm.nih.gov/12221344/">improve</a> neck function – such as manual therapy, exercise and education – can <a href="https://pubmed.ncbi.nlm.nih.gov/27497890/">provide</a> <a href="https://pubmed.ncbi.nlm.nih.gov/38902195/">short- and long-term benefits</a>.</p> <h2>Can physio help with migraine?</h2> <p>Migraine is a neurological disorder whereby the brain has difficulty processing sensory input.</p> <p>This can cause episodic attacks of moderate to severe headache, as well as:</p> <ul> <li>sensitivity to light and noise</li> <li>nausea and</li> <li>intolerance to physical exertion.</li> </ul> <p>There are many triggers. Everyone’s are different and identifying yours is crucial to self-management of migraine. Medication can also help, so seeing a GP is the first step if you <a href="https://headacheaustralia.org.au/migraine/">suspect you have migraine</a>.</p> <p>About <a href="https://pubmed.ncbi.nlm.nih.gov/35166137/">70-80%</a> of people with migraine also have neck pain, commonly just before or at the onset of a migraine attack. This can make people think their neck pain is triggering the migraine.</p> <p>While this may be true in some people, our <a href="https://pubmed.ncbi.nlm.nih.gov/34214181/">research</a> has shown many people with migraine have nothing wrong with their neck despite having neck pain.</p> <p>In those cases, neck pain is part of migraine and can be a warning (but not a cause or trigger) of an imminent migraine attack. It can signal patients need to take steps to prevent the attack.</p> <p>On the other hand, if the person has musculoskeletal neck disorder, physiotherapy neck treatments may help improve their migraine. Musculoskeletal neck disorder is what physiotherapists call typical neck pain caused by, for instance, a sports injury or sleeping in a weird way.</p> <p>You may have heard of the Watson manual therapy technique being used to treat migraine. It involves applying manual pressure to the upper cervical spine and neck area.</p> <p>There are currently no peer-reviewed studies looking at how effective this technique is for migraine.</p> <p>However, recent studies investigating a combination of manual therapy, neck exercises and education tailored to the individual’s circumstances <a href="https://pubmed.ncbi.nlm.nih.gov/36739510/">show</a> some <a href="https://www.researchgate.net/publication/342117351_Neck_treatment_compared_to_aerobic_exercise_in_migraine_A_preference-based_clinical_trial">small effects</a> in improving the number of migraine attacks and the disabling effects of headache.</p> <p>Manual therapy and neck exercises can also give short-term pain relief.</p> <p>However, in some cases the neck can become very sensitive and easily aggravated in <a href="https://pubmed.ncbi.nlm.nih.gov/15907257/">migraine</a>. That means inappropriate assessment or treatment could end up triggering a migraine.</p> <p>Physiotherapy can help with migraine but you first need a comprehensive and skilled physical assessment of the neck by an experienced physiotherapist. It’s crucial to identify if a musculoskeletal neck disorder is present and, if so, which type of neck treatment is needed.</p> <p>It is also important people with migraine understand how their migraine is triggered, what lifestyle factors contribute to it and when to take the appropriate medications to help manage their migraines.</p> <p>A trained physiotherapist can provide some of this information and help patients make sense of their condition and recommend the patient see their GP for medication, when appropriate.</p> <h2>What about tension headaches?</h2> <p>Tension type headache is the most common type of headache, characterised by a feeling of “tightness” or “band-like” pain around the head.</p> <p>Nausea and sensitivity to light and noise are not usually present with this type of headache.</p> <p>Like migraine, tension type headache is often associated with neck pain and also has different aggravating factors, not all of which are due to the neck.</p> <p>Again, a detailed assessment by a trained physiotherapist is needed to identify if the neck is involved and what type of neck treatment is best.</p> <p>There is some evidence a combination of <a href="https://pubmed.ncbi.nlm.nih.gov/35236143/">manual therapy and exercise</a> can reduce tension type headache.</p> <p>Physiotherapists can also provide education and advice on aggravating factors and self management.</p> <h2>Seeking help</h2> <p>There are many types and causes of headache. If you suffer frequent headaches or have a new or unusual headache, ask a doctor to investigate.</p> <p>There is good evidence physiotherapy treatment will improve cervicogenic headache and emerging evidence it might help migraine and tension type headache (alongside usual medical care).</p> <p>If you are wondering if you have cervicogenic headache or if you have bothersome neck pain associated with headache, ask your doctor to refer you to a skilled physiotherapist trained in headache treatment. A careful assessment can determine if physiotherapy treatment will help.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/256581/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By </em><a href="https://theconversation.com/profiles/zhiqi-liang-2392952"><em>Zhiqi Liang</em></a><em>, Lecturer in Physiotherapy, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a>; <a href="https://theconversation.com/profiles/julia-treleaven-2393094">Julia Treleaven</a>, Associate Professor in Physiotherapy, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a>, and <a href="https://theconversation.com/profiles/lucy-thomas-2392963">Lucy Thomas</a>, Teaching and research academic in Physiotherapy, <a href="https://theconversation.com/institutions/the-university-of-queensland-805">The University of Queensland</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/can-you-treat-headaches-with-physiotherapy-heres-what-the-research-says-256581">original article</a>.</em></p> <p><em>Image: Marcus Aurelius / Pexels</em></p> </div>

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Alan Alda shares health update on battle with Parkinson's

<p>Beloved TV icon Alan Alda has shared a candid and characteristically humorous update on his ongoing battle with Parkinson’s disease, a condition he was diagnosed with in 2015.</p> <p>“Making progress,” Alda, 89, quipped to <em>People</em>. “I didn’t say in which direction.”</p> <p>The Emmy-winning actor, best known for his role as Hawkeye Pierce on the long-running hit series <em>MASH</em>, continues to embrace his trademark wit and optimism, even as the neurological disease increasingly shapes his daily life.</p> <p>Alda described the progression of Parkinson’s as turning what was once a “part-time job” into “almost a full-time job”, yet he remains determined to find joy and humour in each day. “It keeps me always looking for the funny side,” he said.</p> <p>Living with Parkinson’s isn't Alda's only neurological challenge – he also has prosopagnosia, or face blindness, which prevents him from recognising even familiar faces. Despite this, Alda maintains a playful spirit, saying each day is about “finding a new way to do something”, likening it to a daily game.</p> <p>Though his health has prompted some lifestyle adjustments, Alda hasn't stepped away from the screen entirely. He recently appeared in Netflix’s new series adaptation of <em>The Four Seasons</em>, a modern reimagining of the 1981 film he originally wrote, directed and starred in. The show, led by Tina Fey, has already been renewed for a second season. Alda appeared in a new role, playing the father of Anne, rather than reprising his original character, the high-strung lawyer Jack.</p> <p>In a video message posted on X (formerly Twitter), Alda congratulated Fey on the show's success. “Last week her new series <em>The Four Seasons</em> was number one on Netflix worldwide,” he said. “Tina, I knew you'd take the movie that I wrote and directed all those years ago and do something wonderful with it – and you have.” </p> <p>Beyond his professional life, Alda continues to celebrate a remarkable personal journey with his wife of 68 years, Arlene, 92. It was Arlene who first noticed an early symptom of Parkinson’s – the absence of Alda’s usual arm swing while walking – a subtle sign of the disease’s characteristic muscle stiffness.</p> <p>Despite the challenges, Alda remains a powerful example of strength, humour and creativity in the face of adversity – a role he seems to play as effortlessly off-screen as he has for decades on it.</p> <p><em>Images: Instagram</em></p>

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"Keen to keep living": Hero professor's heartbreaking health update

<p>Professor Richard Scolyer, the <span style="font-family: -apple-system, BlinkMacSystemFont, 'Segoe UI', Roboto, Oxygen, Ubuntu, Cantarell, 'Open Sans', 'Helvetica Neue', sans-serif;">acclaimed melanoma researcher and former Australian of the Year,</span><span style="font-family: -apple-system, BlinkMacSystemFont, 'Segoe UI', Roboto, Oxygen, Ubuntu, Cantarell, 'Open Sans', 'Helvetica Neue', sans-serif;"> has revealed a devastating update in his ongoing battle with brain cancer, announcing that his aggressive glioblastoma has progressed once again.</span></p> <p>Professor Scolyer, 58, was diagnosed in 2023 with the terminal form of brain cancer and initially given just eight months to live. Defying the odds, he has endured 18 months of life thanks to a groundbreaking experimental immunotherapy based on melanoma research – a treatment approach that has drawn global attention.</p> <p>However, in a candid social media post on Monday, Professor Scolyer confirmed that a recent MRI had shown further tumour growth on the left side of his brain.</p> <p>“While this may not be the best direction to be heading with my changes, amazingly (to me), I still seem keen to keep living, loving and having fun, whenever possible,” he wrote. “I feel like there are quite a few people on my team (including my family & friends) and they make me happy and proud.”</p> <p>Jointly named the 2024 Australian of the Year with fellow Melanoma Institute Australia co-director Professor Georgina Long, Scolyer has become known not only for his scientific leadership but for his openness and optimism in facing a terminal diagnosis.</p> <p>The most recent setback follows a recurrence of the cancer earlier this year, which led to surgery in March. While doctors were able to remove much of the tumour, Scolyer explained that microscopic “tentacles” remain, requiring further treatment.</p> <p>“Depending on what the scan shows … that will help choose what are the next forms of therapy that I can have to see where we need to go from here,” he said in a previous update.</p> <p>Despite ongoing physical and emotional challenges, Scolyer continues to cherish time with his wife, Katie, and their children.</p> <p>“Sometimes I’m happy to have fun, but some of the therapies have knocked me around a bit, so I can’t do some of the things I love doing,” he said. “I’m still having a fun time at home with my kids … and my beautiful wife Katie has been using her incredible intellect to help me speak to different doctors about various options.”</p> <blockquote class="instagram-media" style="background: #FFF; border: 0; border-radius: 3px; box-shadow: 0 0 1px 0 rgba(0,0,0,0.5),0 1px 10px 0 rgba(0,0,0,0.15); margin: 1px; max-width: 540px; min-width: 326px; padding: 0; width: calc(100% - 2px);" data-instgrm-captioned="" data-instgrm-permalink="https://www.instagram.com/p/DJ1JNz9zW8W/?utm_source=ig_embed&utm_campaign=loading" data-instgrm-version="14"> <div style="padding: 16px;"> <div style="display: flex; flex-direction: row; align-items: center;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 40px; margin-right: 14px; width: 40px;"> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 100px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 60px;"> </div> </div> </div> <div style="padding: 19% 0;"> </div> <div style="display: block; height: 50px; margin: 0 auto 12px; width: 50px;"> </div> <div style="padding-top: 8px;"> <div style="color: #3897f0; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: 550; line-height: 18px;">View this post on Instagram</div> </div> <div style="padding: 12.5% 0;"> </div> <div style="display: flex; flex-direction: row; margin-bottom: 14px; align-items: center;"> <div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(0px) translateY(7px);"> </div> <div style="background-color: #f4f4f4; height: 12.5px; transform: rotate(-45deg) translateX(3px) translateY(1px); width: 12.5px; flex-grow: 0; margin-right: 14px; margin-left: 2px;"> </div> <div style="background-color: #f4f4f4; border-radius: 50%; height: 12.5px; width: 12.5px; transform: translateX(9px) translateY(-18px);"> </div> </div> <div style="margin-left: 8px;"> <div style="background-color: #f4f4f4; border-radius: 50%; flex-grow: 0; height: 20px; width: 20px;"> </div> <div style="width: 0; height: 0; border-top: 2px solid transparent; border-left: 6px solid #f4f4f4; border-bottom: 2px solid transparent; transform: translateX(16px) translateY(-4px) rotate(30deg);"> </div> </div> <div style="margin-left: auto;"> <div style="width: 0px; border-top: 8px solid #F4F4F4; border-right: 8px solid transparent; transform: translateY(16px);"> </div> <div style="background-color: #f4f4f4; flex-grow: 0; height: 12px; width: 16px; transform: translateY(-4px);"> </div> <div style="width: 0; height: 0; border-top: 8px solid #F4F4F4; border-left: 8px solid transparent; transform: translateY(-4px) translateX(8px);"> </div> </div> </div> <div style="display: flex; flex-direction: column; flex-grow: 1; justify-content: center; margin-bottom: 24px;"> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; margin-bottom: 6px; width: 224px;"> </div> <div style="background-color: #f4f4f4; border-radius: 4px; flex-grow: 0; height: 14px; width: 144px;"> </div> </div> <p style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; line-height: 17px; margin-bottom: 0; margin-top: 8px; overflow: hidden; padding: 8px 0 7px; text-align: center; text-overflow: ellipsis; white-space: nowrap;"><a style="color: #c9c8cd; font-family: Arial,sans-serif; font-size: 14px; font-style: normal; font-weight: normal; line-height: 17px; text-decoration: none;" href="https://www.instagram.com/p/DJ1JNz9zW8W/?utm_source=ig_embed&utm_campaign=loading" target="_blank" rel="noopener">A post shared by Prof Richard Scolyer AO - My Uncertain Path (@profrscolyer)</a></p> </div> </blockquote> <p>Looking ahead, Professor Scolyer anticipates another operation and remains hopeful that ongoing research and innovation may unlock new options for glioblastoma patients worldwide.</p> <p>“Fingers crossed this operation isn’t so bad and we can move forward with the next form of therapy,” he said, “and hopefully push things along faster to try and get things open up for many, many patients who have got glioblastoma.”</p> <p><em>Images: Instagram</em></p>

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Lisa Curry’s emotional plea 5 years after Jaimi's tragic death

<p>Aussie swimming legend Lisa Curry has made a heartfelt appeal in memory of her late daughter, Jaimi Kenny, who tragically passed away in 2020 at the age of 33 following a prolonged and painful battle with alcoholism and an eating disorder.</p> <p>Appearing on Nine News, the 63-year-old former Olympian opened up about her devastating loss and called on Australians to participate in a groundbreaking new genetic study that aims to detect eating disorders from birth.</p> <p>“She just sort of took a breath and then her hands went cold – that was it,” Lisa recalled, her voice heavy with emotion.</p> <p>Jaimi’s struggle with anorexia, bulimia and binge eating left her mother with more questions than answers. “She’d say, ‘Where did it come from? Why am I like this?’” Lisa shared, still haunted by her daughter’s anguish.</p> <p>Now, in the midst of grief, Lisa is channelling her pain into purpose. She is urging Australians to volunteer for a pioneering genetic research project led by Queensland’s QIMR Berghofer Medical Research Institute. The study hopes to enlist 4,000 local participants and 100,000 globally, collecting saliva samples to identify potential genetic markers linked to eating disorders.</p> <p>Professor Nick Martin, who is leading the study, explained that genetic predisposition plays a significant role in the development of such illnesses – a discovery that could change lives and save them.</p> <p>Lisa’s renewed call for awareness and action follows her own harrowing health battles. In recent months, she revealed she’s been in “recovery” from the unrelenting grief of losing Jaimi. After more than three years, she finally returned to the gym – a milestone made even more significant by the physical pain of a hip replacement, the result of a fall while husky sledding in Canada last year.</p> <p>Lisa’s health history also includes myocarditis, a mitral valve prolapse inherited from her mother, and a fractured sternum. "Seventeen years ago, I had 22,000 irregular heartbeats a day,” she wrote. “That was the start of a different life.”</p> <p>Since the loss of Jaimi, Lisa has committed herself to raising awareness about the silent struggles so many face – especially when it comes to eating disorders. Her message is simple but urgent: more research, more understanding and more compassion are desperately needed.</p> <p><em>If you or someone you know is struggling with an eating disorder, support is available. Contact the Butterfly Foundation’s national helpline at 1800 33 4673 or visit <a href="https://butterfly.org.au/" target="_blank" rel="noopener">thebutterflyfoundation.org.au</a>.</em></p> <p><em>Images: Instagram / Nine News</em></p>

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More people are trying medicinal cannabis for chronic pain. But does it work?

<div class="theconversation-article-body"> <p>More Australians than ever are being prescribed <a href="https://www.betterhealth.vic.gov.au/health/conditionsandtreatments/medicinal-cannabis">medicinal cannabis</a>.</p> <p>Medicinal cannabis refers to legally prescribed cannabis products. These are either the plant itself, or naturally occurring ingredients extracted from the plant. These ingredients, such as THC (tetrahydrocannabinol) and CBD (cannabidiol), are called cannabinoids. Some cannabinoids are also made in labs to act like the ones in the plant.</p> <p>Medicinal cannabis comes in different forms, such as oils, capsules, dried flower (used in a vaporiser), sprays and edible forms such as gummies.</p> <p>Since regulatory changes in 2016 made medicinal cannabis more accessible, Australia’s regulator has issued <a href="https://dashboard-data.health.gov.au/single/?appid=1066afbe-2b37-427d-8c47-2caa5082cccc&sheet=088f611b-10de-4d72-be68-ccf8d12c54e9&select=clearall">more than 700,000 approvals</a>. (But approvals for medicinal cannabis don’t reflect the actual number of patients treated. One patient may have multiple approvals, and not all approved products are necessarily prescribed or supplied.)</p> <p>Around half of the approvals have been for chronic pain that isn’t caused by cancer.</p> <p>In Australia, chronic pain affects around <a href="https://www.aihw.gov.au/reports/chronic-disease/chronic-pain-in-australia/summary">one in five</a> Australians aged 45 and over, with an enormous impact on people’s lives.</p> <p>So what does the current evidence tell us about the effectiveness of medicinal cannabis for chronic pain?</p> <h2>What the evidence shows</h2> <p>A <a href="https://www.bmj.com/content/374/bmj.n1034">2021 review of 32 randomised controlled trials</a> involving nearly 5,200 people with chronic pain, examined the effects of medicinal cannabis or cannabinoids. The study found a small improvements in pain and physical functioning compared with a placebo.</p> <p>A <a href="https://pubmed.ncbi.nlm.nih.gov/29847469/">previous review</a> found that to achieve a 30% reduction in pain for one person, 24 people would need to be treated with medicinal cannabis.</p> <p>The 2021 review also <a href="https://www.bmj.com/content/374/bmj.n1034">found</a> small improvements in sleep, and no consistent benefits for other quality of life measures, consistent with <a href="https://pubmed.ncbi.nlm.nih.gov/29847469/">previous reviews</a>.</p> <p>This doesn’t mean medicinal cannabis doesn’t help anyone. But it suggests that, on average, the benefits are limited to a smaller number of people.</p> <p>Many pain specialists have <a href="https://theconversation.com/medicinal-cannabis-to-manage-chronic-pain-we-dont-have-evidence-it-works-157324">questioned</a> if the evidence for medicinal cannabis is sufficient to support its use for pain.</p> <p>The <a href="https://www.choosingwisely.org.au/recommendations/fpm6">Faculty of Pain Medicine</a>, the professional body dedicated to the training and education of specialist pain physicians, recommends medical cannabis should be limited to clinical trials.</p> <h2>What does the regulator say?</h2> <p>Guidance from Australia’s regulator, the <a href="https://www.tga.gov.au/products/unapproved-therapeutic-goods/medicinal-cannabis-hub/medicinal-cannabis-guidance-documents/guidance-use-medicinal-cannabis-treatment-chronic-non-cancer-pain-australia">Therapeutic Goods Administration</a> (TGA), on medicinal cannabis for chronic non-cancer pain reflects these uncertainties.</p> <p>The TGA states there is limited evidence medicinal cannabis provides clinically significant pain relief for many pain conditions. Therefore, the potential benefits versus harms should be considered patient-by-patient.</p> <p>The TGA says medicinal cannabis should only be trialled when other standard therapies have been tried and did not provide enough pain relief.</p> <p>In terms of which type of medical cannabis product to use, due to concerns about the safety of inhaled cannabis, the TGA considers pharmaceutical-grade products (such as nabiximols or extracts containing THC and/or CBD) to be safer.</p> <h2>What about people who say it helps?</h2> <p>This evidence may feel at odds with the experiences of people who report relief from medicinal cannabis.</p> <p>In clinical practice, it’s common for individuals to respond differently based on their health conditions, beliefs and many other factors. What works well for one person may not work for another.</p> <p>Research helps us understand what outcomes are typical or expected for most people, but there is variation. Some people may find medicinal cannabis improves their pain, sleep or general well-being – especially if other treatments haven’t helped.</p> <h2>What are the side effects and risks?</h2> <p>Like any medicine, medicinal cannabis has <a href="https://www.tga.gov.au/products/unapproved-therapeutic-goods/medicinal-cannabis-hub/medicinal-cannabis-guidance-documents/guidance-use-medicinal-cannabis-treatment-chronic-non-cancer-pain-australia#s9">potential side effects</a>. These are usually mild to moderate, including drowsiness or sedation, dizziness, impaired concentration, a dry mouth, nausea and cognitive slowing.</p> <p>These side effects are often greater with higher-potency THC products. These are becoming more common on the Australian market. High-potency THC products represent <a href="https://dashboard-data.health.gov.au/single/?appid=1066afbe-2b37-427d-8c47-2caa5082cccc&sheet=088f611b-10de-4d72-be68-ccf8d12c54e9&select=clearall">more than half of approvals in 2025</a>.</p> <p>In research studies, generally <a href="https://pubmed.ncbi.nlm.nih.gov/29847469/">more people experience side effects</a> than report benefits from medical cannabis.</p> <p>Medical cannabis can also <a href="https://pubmed.ncbi.nlm.nih.gov/36317739/">interact with other medications</a>, especially those that cause drowsiness (such as opioids), medicines for mental illness, anti-epileptics, blood thinners and immunosuppressants.</p> <p>Even cannabidiol (CBD), which isn’t considered intoxicating like THC, has been linked to serious drug interactions.</p> <p>These risks are greater when cannabis is prescribed by a doctor who doesn’t regularly manage the patient’s chronic pain or isn’t in contact with their other health-care providers. Since medicinal cannabis is often prescribed through separate telehealth clinics, this fragmented care may increase the risk of harmful interactions.</p> <p>Another concern is developing cannabis use disorder (commonly understood as “addiction”). A 2024 study found <a href="https://www.sciencedirect.com/science/article/pii/S0376871624001844?via%3Dihub">one in four people</a> using medical cannabis develop a cannabis use disorder. Withdrawal symptoms – such as irritability, sleep problems, or cravings – can occur with frequent and heavy use.</p> <p>For some people, tolerance can also develop with long-term use, meaning you need to take higher doses to get the same effect. This can increase the risk of developing a cannabis use disorder.</p> <h2>How does it compare to other treatments?</h2> <p>Like many <a href="https://www.healthdirect.gov.au/chronic-pain">medicines for chronic pain</a>, the effectiveness of medicinal cannabis is modest, and is not recommended as a sole treatment.</p> <p>There’s good evidence that, for conditions like back pain, interventions such as exercise, cognitive behavioural therapy and pain self-management education can <a href="https://theconversation.com/do-any-non-drug-treatments-help-back-pain-heres-what-the-evidence-says-253122">help</a> and may have fewer risks than many medicines.</p> <p>But there are challenges with how accessible and affordable these treatments are for many Australians, <a href="https://www1.racgp.org.au/getattachment/e0603085-695c-4fbb-b7d9-ba77057e5a97/Management-of-chronic-pain-in-a-rural-Australian-s.aspx">especially outside major cities</a>.</p> <h2>So where does this leave patients?</h2> <p>The growing use of medicinal cannabis for chronic pain reflects both a high burden of pain in the community and gaps in access to effective care. While some patients report benefits, the current evidence suggests these are likely to be small for most people, and must be weighed against the risks.</p> <p>If you are considering medicinal cannabis, it’s important to talk to your usual health-care provider, ideally one familiar with your full medical history, to help you decide the best approaches to help manage your pain.<!-- Below is The Conversation's page counter tag. Please DO NOT REMOVE. --><img style="border: none !important; box-shadow: none !important; margin: 0 !important; max-height: 1px !important; max-width: 1px !important; min-height: 1px !important; min-width: 1px !important; opacity: 0 !important; outline: none !important; padding: 0 !important;" src="https://counter.theconversation.com/content/256471/count.gif?distributor=republish-lightbox-basic" alt="The Conversation" width="1" height="1" /><!-- End of code. If you don't see any code above, please get new code from the Advanced tab after you click the republish button. The page counter does not collect any personal data. More info: https://theconversation.com/republishing-guidelines --></p> <p><em>By <a href="https://theconversation.com/profiles/suzanne-nielsen-35849">Suzanne Nielsen</a>, Professor and Deputy Director, Monash Addiction Research Centre, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a> and <a href="https://theconversation.com/profiles/myfanwy-graham-2392855">Myfanwy Graham</a>, NHMRC Postgraduate Scholar and Fulbright Alumna in Public Health Policy, <a href="https://theconversation.com/institutions/monash-university-1065">Monash University</a></em></p> <p><em>This article is republished from <a href="https://theconversation.com">The Conversation</a> under a Creative Commons license. Read the <a href="https://theconversation.com/more-people-are-trying-medicinal-cannabis-for-chronic-pain-but-does-it-work-256471">original article</a>.</em></p> <p><em>Image: </em></p> </div>

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